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Disclosure and Acknowledgment This letter was conceived, written and edited by the authors who take full responsibility for all statements. The authors would like to acknowledge and thank ACUMED for coordination and editorial support during the preparation of this article. ACUMED's contribution was funded by Novartis Pharma AG and
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1.70.6 L 6123% pred ; . Both FEV1 and FVC remained unchanged throughout the study. Blood haemoglobin baseline mean 137.89.7 g?L-1 ; or serum alanine aminotransferase baseline mean 17.7 6.0 U?L-1 ; were within reference ranges and did not change during the trial. No adverse drug effects were observed or reported. The mean Pa, CO2 was 5.40.6 kPa at baseline and decreased on average by 0.80.3 kPa with MPA pv0.001 ; . The mean oxygen tension in arterial blood Pa, CO2 ; was 1.31.6 and did not change significantly during the study period. At baseline, the mean pH was 7.410.03, the change with MPA 0.010.03 ; was not significant. At baseline, the mean base excess BE ; was 0.61.9 mmol?L-1 and the mean HCO3was 25.11.6 mmol?L-1. With MPA, BE decreased by 2.21.2 mmol?L-1 pv0.0001 ; and HCO3- by 1.9 1.0 mmol?L-1 from baseline pv0.0001 ; . The mean concentrations of serum leptin fig. 2 ; or NPY fig. 3 ; did not change. At baseline, mean serum leptin concentration was 19.89.9 mg?L-1. On MPA, leptin was 19.79.8 mg?L-1. At baseline, mean serum NPY concentration was 94.018.3 pmol?L-1. On MPA, NPY was 85.141.2 pmol?L-1. When treated as a single-group placebo-controlled trial, the level of significance further increased the change in Pa, CO2 p 0.0001 ; , while that for Pa, O2 and
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