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Brook, DW et al. Drug use and the risk of major depressive disorder, alcohol dependence, and substance use disorders. Archives of General Psychiatry, 59: 1039-1044, 2002. This longitudinal research of comorbid disorders found that early marijuana use during childhood and adolescence increased the risk of major depression by 17 percent. This study called attention to the importance of the psychiatric implications of early drug use. Fergusson, DM et al. Cannabis use and psychosocial adjustment in adolescence and young adulthood. Addiction, 97: 1123-1135, 2002. This study of 1, 265 New Zealand children over a 21-year period found that marijuana use, particularly heavy or regular use, was associated with later increases in depression, suicidal thoughts and suicide attempts. Greenblatt, J. Adolescent self-reported behaviors and their association with marijuana use. Based on data from the National Household Survey on Drug Abuse, 1994-1996, SAMSHA, 1998. This research shows that kids age 12 to 17 who smoke marijuana weekly are three times more likely than non-users to have thoughts about committing suicide. Lynskey, M et al. Major depressive disorder, suicidal ideation, and suicide attempt in twins discordant for cannabis dependence and early-onset cannabis use. Archives of General Psychiatry, 61: 1026-1032, 2004. This study looked at 600 same-sex twins, one of whom was dependent upon marijuana and one of whom was not. It found that the twin who was dependent on marijuana was almost three times more likely to think about suicide and attempt suicide than his her non-marijuana dependent cotwin. Additionally, cannabis dependence was associated with higher risk of major depressive disorder in fraternal but not in identical twins. Patton, GC et al. Cannabis use and mental health in young people: cohort study. British Medical Journal, 325: 1195-1198, 2002. In this study, daily use of marijuana among girls increased the risk of depression five times. Weekly or more frequent marijuana use in teenagers doubled the risk of depression and anxiety. Ramstrom, J. Adverse Health Consequences of Cannabis Use: A survey of scientific studies published up to and including the autumn of 2003. National Institute of Public Health, Sweden, 2004. This is an extensive literature review of studies conducted worldwide on the detrimental effects of marijuana. It is an update of a review initially published in 1996 and covers studies through the fall of 2003. It finds a link between marijuana and depression and suicidal tendencies. The author points out that there is a growing body of evidence to support the claim that cannabis can provoke schizophrenia and motrin. Consult the prescribing doctor and read the package insert before using these or any other medications or supplements.

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9.0 CI, 1.6-50.1 ; with CEE plus daily MPA and 7.2 CI, 1.3-40.0 ; with CEE plus MP compared to CEE alone. The placebo group had no density grade increases. These and our results indicate that MPA may enhance the mitotic rate of normal epithelial breast cells in the presence of growth factors and therefore long-term use may increase the probability of faults in DNA-replication. Furthermore, the results of the WHI indicated that patients who were not using hormones prior to the start of the study had no increased hazard ratio for breast cancer, however subjects with prior hormone use for up to five, five to ten and more than 10 years showed an increasing risk Writing Group for the Women's Health Initiative Investigators 2002 ; . These data suggest that long-term use of MPA may increase breast cancer risk by enhancing the mitotic rate of normal epithelial cells. CMA has been shown to have no influence on breast cancer risk in a casecontrol study involving combined oral contraceptives containing CMA. Risk was not elevated in ever-users, did not increase with duration of use and did not change with time since initial exposure or with time since most recent use Ebeling et al 1991 ; . In contrast to this, another study of the use of oral contraceptives containing CMA found an elevated relative risk for breast cancer of 1.3. The authors of this study state that although not statistically significant, the positive correlation observed for breast cancer may be real Nischan and Ebeling 1984 ; , which is consistent with our stimulatory results. Up to now, there is a paucity of data available regarding the effects of CMA on the proliferation of normal epithelial breast cells. P in combination with estrogen has been shown to increase healthy breast epithelial cell proliferation when injected daily into ovariectomized mice for 56 days Rafaat et al 2001 ; . In contrast, in a small clinical trial involving 40 postmenopausal women, exposure to P for 14 days reduced the E2-induced proliferation of normal breast cells in vivo Foidart et al 1998 ; . In their study of women receiving exclusively or mostly a transdermal E2 gel and a progestogen other than MPA oral micronized P was used in 58% of patients, MPA in less.

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